![]() Instead of clicking the Search button, just press Enter. If you are unable to add a bookmarklet in Mozilla Firefox according to the instructions above, there is another way right click on a link and select Bookmark this linkā¦ Now you can drag this link from Bookmarks to the Bookmarks Toolbar. If you want to type a character which isn't on your keyboard, simply pick it from a list of special characters. A bookmarklet is a small JavaScript code stored as a bookmark in your browser. There is a way to enable word translation from any page: Bookmarklets. Afterwards, you simply type the chosen keyword in the address bar to start the search in the chosen dictionary. In Chrome, first click on a language pair and change the search keyword in the field 'Keyword' to a keyword (eg: 'eudict'). And you're ready to go select EUdict from the drop-down list in search field (Firefox) or address bar (IE), input a word and press Enter. To add EUdict alongside Google, Yahoo!, Amazon and other search engines in Mozilla Firefox or Internet Explorer, simply click on link after the title Browser integration, select appropriate language pair and confirm your decision. Perhaps the best way to enable dictionary search is through integration into the search field of your browser. For the same reason the Chinese dictionary contains traditional and simplified Chinese terms on one side and Pinyin and English terms on the other. ![]() There are two Japanese-English (and Japanese-French) dictionaries and one contains Kanji and Kana (Kana in English and French pair due to improved searching). Look at the complete list of languages: Available language pairs The most common way is by word input (you must know which language the word is in) but you can also use your browser's search box and bookmarklets (or favelets). There are several ways to use this dictionary. Total number of translations (in millions): 15 Please help us improve this site by translating its interface. Therefore, macrophages, via their products of secretion acting on 5'-nucleotidase, could modulate adenosine production in the glomerular capillaries.Esperanto is only partially translated. Taken together, these results indicate that IL-1 beta, essentially, and TNF-alpha, to a lesser extent, regulate 5'-nucleotidase expression in the plasma membrane of cultured mesangial cells and that their effect depends in part on PGE2 synthesis. In addition, PGE2 itself produced a dose-related (0.1-10 microM) increase of 5'-nucleotidase activity with a maximum of 2.2 times basal value. Their inhibitory effect was abolished in the presence of prostaglandin E2 (PGE2). Cyclo-oxygenase inhibitors such as indomethacin and ibuprofen inhibited approximately 50% of the effects of both cytokines. Cycloheximide, a blocker of protein synthesis, suppressed the cytokine-dependent increase of 5'-nucleotidase activity. Stimulation of 5'-nucleotidase by IL-1 beta and TNF-alpha was specific since the activity of other ectoenzymes, such as Mg2(+)-ATPase, was unchanged. The effects of both cytokines were additive. Maximum increases reached 4.5 times and 1.7 times basal values for IL-1 beta (20 U/ml) and TNF-alpha (25 ng/ml), respectively. IL-1 beta and TNF-alpha stimulated mesangial cell 5'-nucleotidase activity in a dose-dependent manner after treatment for 24 hr. ![]() Because ecto-5'-nucleotidase activity of rat glomerular mesangial cells has been shown to increase upon interaction with macrophages in vitro, it was examined whether interleukin-1 beta (IL-1 beta) and tumour necrosis factor-alpha (TNF-alpha), two macrophage-released cytokines, were responsible for this effect.
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